Gastric volvulus occurs when a congenital or acquired abnormality
allows the stomach to twist upon itself. Some known causes of
gastric volvulus include: abnormal ligamentous connections,
congenital diaphragmatic defects, gastric tumors, ulcers,
organomegaly, intra-abdominal masses, elevation of the left
hemidiaphragm, phrenic nerve paralysis, and overeating. We report
the first patient, to our knowledge, to develop a gastric volvulus
secondary to incarcerated inguinal hernia.
An unresponsive 83-year-old white male presented with respiratory
failure, hypotension, and marked abdominal distention. There was no
history of vomiting. Abdominal exam revealed marked distention and
hypoactive bowel sounds. Initial labs included a Hbg of 13.2 gm/dl,
WBC 15.7/cmmm, platelets 149,000/cmm, pH 7.11, HCO3 10 mmol/L and
lactic acid 10.0 mmol/L. Attempts to place a nasogastric tube were
unsuccessful. An x-ray showed massive dilation of the stomach and
multiple loops of small bowel. The nasogastric tube was coiled
within the esophagus.
An emergent upper GI endoscopy revealed a dilated esophagus with
a large amount of residual fluid. Near the GE junction, a complete
obstruction due to esophageal torsion was noted. The endoscope was
unable to pass the point of torsion.
An exploratory laparotomy revealed an ischemic, massively dilated
stomach, twisted around its organoaxial axis and incarcerated
jejunum through a right inguinal hernia. Six liters of feculent
gastric fluid were aspirated through a gastrotomy, and the stomach
was untwisted. The incarcerated jejunum was reduced, the hernia
repaired, and a gastrostomy tube was placed. Postoperatively, the
patient rapidly deteriorated and died 12 hours later due to
multi-system organ failure and profound metabolic acidosis.
Many causes of gastric volvulus have been previously described.
Incarcerated inguinal hernia should be included in the list of
possible etiologies of gastric volvulus.